Bodun Ma
Neurology researcher in Suzhou and first author of a 2026 study on how the enzyme PIP4K2A protects the brain after stroke by restoring autophagy through AKT/mTOR
Department of Neurology, The Affiliated Suzhou Hospital of Nanjing Medical University (Suzhou Municipal Hospital), Gusu School, Nanjing Medical University (at the time of the study, 2026)
Restoring blood flow after a stroke saves brain tissue, but the sudden return of oxygen also injures cells. Bodun Ma's 2026 study, with Hongxuan Feng as senior author, asked whether the lipid kinase PIP4K2A could limit this reperfusion damage.
In the team's experimental models, more PIP4K2A meant less injury. Two changes went with it: less of a TRIB3-p62 complex that can jam autophagy, the cell's recycling pathway, and altered AKT/mTOR signalling, which let autophagy flow again. mTOR normally holds autophagy back when growth signals are high, so tuning this pathway changes how well damaged neurons clear debris. These are laboratory findings, not results in stroke patients.
PubMed gives the Department of Neurology at the Affiliated Suzhou Hospital of Nanjing Medical University as Bodun Ma's affiliation. A current position could not be confirmed from an official page.
Milestones in the Atlas
A selection, not a full bibliography: only papers that meet the Atlas inclusion criteria.
| Year | Evidence | Study |
|---|---|---|
| 2026 | A | PIP4K2A Attenuates Cerebral Ischemia/Reperfusion Injury by Reducing the TRIB3-p62 Complex Burden and Modulating AKT/mTOR Signaling. MA2026 First author: showed that PIP4K2A overexpression reduces ischemia/reperfusion brain injury by lowering the TRIB3-p62 burden and modulating AKT/mTOR to restore autophagic flux. |
Co-authors in the Atlas 4
People with a profile here who share at least one study with Bodun Ma.
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- Hongxuan Feng Neurologist at Suzhou Municipal Hospital and senior author of a 2026 stroke study proposing PIP4K2A as a protector of brain tissue through AKT/mTOR signalling and autophagy
- Mei Li Scientist at the Pediatrics Research Institute of Children's Hospital of Soochow University studying brain injury, who contributed to a study of how PIP4K2A protects the brain after stroke
- Shuai Yu Neurology researcher who contributed to a study showing that the lipid kinase PIP4K2A protects the brain after ischaemic stroke by relieving a block on autophagy and boosting AKT/mTOR signalling
- Xiaotian Yu Stroke neurology researcher who contributed to a study of how PIP4K2A limits brain injury after ischaemia and reperfusion through the TRIB3–p62 and AKT–mTOR pathways
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