Ling Zhang
Contributed to a 2026 toxicology study linking early-life exposure to microplastics and the plasticiser DEHP to autism-relevant brain changes through impaired mTOR-regulated autophagy
School of Public Health, Hubei Province Key Laboratory of Occupational Hazard Identification and Control · Wuhan University of Science and Technology, China
Ling Zhang is part of a collaboration (with a Central China Normal University toxicology group) on a 2026 paper exposing young mice to food-grade polypropylene microplastics and the plasticizer DEHP. The combined exposure produced autism-spectrum-disorder-relevant behavioral and synaptic changes, traced to activation of mTOR signaling and impaired autophagy — partially reversed by the mTOR inhibitor rapamycin.
Milestones in the Atlas
A selection, not a full bibliography: only papers that meet the Atlas inclusion criteria.
| Year | Evidence | Study |
|---|---|---|
| 2026 | A | Early-life exposure to polypropylene microplastics and DEHP induces ASD-relevant neurodevelopmental alterations involving mTOR-regulated autophagy impairment YANG2026 Co-author on the finding that early-life polypropylene-microplastic and DEHP co-exposure drives ASD-relevant neurodevelopmental damage in mice through mTOR-pathway activation and autophagy impairment. |
Co-authors in the Atlas 4
People with a profile here who share at least one study with Ling Zhang.
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- Ge Yang Researcher in Rui Li's group at Central China Normal University who has led mouse studies on how microplastics, nanoplastics and the plasticiser DEHP affect the young brain
- Rui Li Toxicologist at Central China Normal University in Wuhan who leads mouse studies of how early-life exposure to microplastics, nanoplastics and plastic additives harms the developing brain
- Xiaojie Sun Contributed to a 2026 toxicology study linking early-life exposure to microplastics and the plasticiser DEHP to autism-relevant brain changes through impaired mTOR-regulated autophagy
- Yifei Wang Environmental toxicologist who contributed to a study of how early-life exposure to food-contact microplastics and a common plasticiser disrupts brain development through mTOR signalling
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