Oliver's mTOR Atlas Evidence Platform
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Leucine

Nutrient/Metabolite · 5 studies in the Atlas

Branched-chain essential amino acid and the best-characterised nutrient input to mTORC1. Free leucine binds Sestrin2, releasing the GATOR2 brake; a competing model has leucyl-tRNA synthetase as the sensor instead.

The amino acid the cell watches most closely.

Binds Sestrin2 with ~20 µM Kd – within the range over which intracellular leucine actually fluctuates, which is the main argument that Sestrin2 is a physiological sensor rather than a binder.

Evidence at a glance

EvidenceWhat it meansStudies
M Molecular — cells, biochemistry, structure5

No direct human evidence in the Atlas for this entity yet — everything below rests on animal or molecular work.

Studies

YearEvidenceStudy
2017 M mTORC1 Activator SLC38A9 Is Required to Efflux Essential Amino Acids from Lysosomes and Use Protein as a Nutrient WYA2017 SLC38A9 effluxes essential amino acids (e.g. leucine) from lysosomes to activate mTORC1.
2015 M Sestrin2 is a leucine sensor for the mTORC1 pathway WOL2015 Sestrin2 is a direct leucine sensor whose leucine binding releases GATOR2 to activate mTORC1.
2015 M Structural basis for leucine sensing by the Sestrin2-mTORC1 pathway SAX2015 Solves the crystal structure of Sestrin2 bound to leucine, revealing the molecular pocket that lets it act as mTORC1's dedicated leucine sensor.
2015 M Metabolism. Differential regulation of mTORC1 by leucine and glutamine JEW2015 Glutamine activates mTORC1 via a Rag-independent, Arf1-dependent route distinct from leucine.
2012 M Leucyl-tRNA synthetase is an intracellular leucine sensor for the mTORC1-signaling pathway HAN2012 Leucyl-tRNA synthetase acts as an intracellular leucine sensor activating mTORC1 via the Rag pathway.

Related entities

LARS (leucyl-tRNA synthetase)GlutamineSestrin2mTORC1 1SLC38A9 1